What side effects can Atenolol have?
Below you will find the most important information about possible known side effects of Atenolol.
These side effects do not occur, but you can. Because every person reacts differently to medication. Please also note that the type and frequency of side effects may vary depending on the drug formulation (eg tablet, syringe, ointment).
Uncommon side effects:
tiredness, headache, drowsiness, dizziness, confusion, hallucinations, blurred vision, sleep disorders, nightmares, depressive moods, increased sweating, sudden drop in blood pressure, short-term unconsciousness, enhancement of existing myocardial insufficiency, slow heartbeat (bradycardia), mild Cardiac arrhythmia, allergic reactions (redness, rashes and itching), discomfort in the hands and feet, enhancement of existing circulatory disorders, impairment of kidney function, gastrointestinal discomfort, diarrhea, constipation, muscle spasms, muscle weakness, reduction of lacrimation (especially in contact lens wearers)! !!!
Rare side effects:
disorders of lipid metabolism, erectile dysfunction, reduction of sex drive, exacerbation of existing complaints by airway constriction, dyspnea, conjunctivitis, dry mouth, worsening of the condition of diabetes mellitus (diabetes mellitus), tendency to hypoglycaemia (especially during exercise) .
Very rare side effects:
appearance or intensification of psoriasis, onset or enhancement of angina pectoris.
During therapy with atenolol kidney and liver function as well as blood count and blood sugar have to be checked at regular intervals.
Atenolol may mask the symptoms of hypoglycaemia in patients with diabetes mellitus and the signs of hyperthyroidism.
What interactions does Atenolol show?
Please note that the interactions may vary depending on the drug formulation of a drug (eg tablet, syringe, ointment).
Hypertension, dehydration, vasodilation, tricyclic antidepressants, barbiturates (for epilepsy) and phenothiazines (for depression) potentiate the antihypertensive effect of atenolol. Calcium antagonists such as nifedipine also have this interaction, but may still lead to the development of cardiac muscle weakness.
Anesthetics and anesthetics increased the blood pressure drop through atenolol. The muscle-relaxing effect of some muscle relaxants such as suxamethonium or tubocurarine is intensified and prolonged by atenolol. Therefore, the anesthetist must always be informed of the use of the active substance before surgery.
Calcium antagonists such as verapamil or diltiazem as well as antiarrhythmics (eg disopyramide) increase the inhibition of cardiac activity by atenolol. It may result in low blood pressure, slow heartbeat, or other cardiac arrhythmias, which requires careful patient monitoring by the physician.
Cardiac glycosides (cardiac insufficiency), the blood pressure reducers reserpine, guanfacine and clonidine, and alpha-methyldopa (in Parkinson’s disease) can inhibit atenolol’s cardiac conduction and dangerously slow the heartbeat. After a sudden stop of treatment with clonidine with concomitant administration of atenolol, the blood pressure may increase excessively. It must therefore first a few days before the administration of atenolol be stopped, then clonidine can be discontinued gradually.
The hypoglycemic effect of oral antidiabetics and insulin (for the treatment of diabetes) is enhanced by atenolol. Signs of hypoglycemia – we may have tachycardia and trembling masked by the drug or be mitigated.
Therefore, regular blood glucose monitoring is required.
Cardiovascular agents such as epinephrine and norepinephrine (alpha and beta sympathomimetics), but also the administration of MAO inhibitors (for depression) can lead to excessive blood pressure increase.
The non-steroidal anti-inflammatory drug indomethacin reduces the hypotensive effect of atenolol.